Alessandro Riberi: un mito della medicina torinese dell’ Dianzani, M U ( Mario Istituzioni di patologia generale Dianzani, M U (Mario Umberto), Dianzani “Istituzioni di Patologia Generale” IV ed – UTET ed. Educational objectives, This course is for the student the first teaching that allows to understand the. Reference texts, -PONTIERI FRATI, RUSSO: Volume I- Patologia Generale. Fisiopatologia Generale Ed. PICCIN -DIANZANI: Istituzioni di Patologia Generale.
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Immunity and tumor growth control.
Study-unit MOLECULAR PATOLOGY – Degree Course: Biotechnology
Mutations in the DNA repair mechanisms. Main knowledge acquired will be: Xeroderma Pigmetosum and Ataxia-telangiectasia. Extended program Hypertrophy and hyperplasia physiological and causes. Disease complex and multifactorial inheritance. Text size Normal Large. Carcinogenicity and co-carcinogenesis, mechanisms of transformation. Amiloidosis, Hyaline, Mucoid and Fibrinoid degenerations. Oncogenes and tumor suppressor genes. Prerequisites In order to be able to understand and apply the majority of argument described within the Course, it is necessary to have successfully passed the Physiology exam.
Istituzioni di Patologia Generale Ed. The exam consists of an oral test as interview of about minutes long aiming to ascertain the knowledge level and the understanding capability acquired by the student on issues proposed.
Identification of causes of disease to give the correct clinical approach of diseasesAnalysis of biomedical information from diagnostic techniques. Plasmatic and tissutal mediators of inflammation.
Overall response of the body to heat and cold. Characteristics of normal and transformed cell. Teaching methods To the students will be given lectures. Local and generalized edema. Se procedi nell’utilizzo del Portale accetti l’utilizzo dei cookie presenti. Oxidative stress and reperfusion injury.
Sono presenti servizi di terze parti Facebook, Twitter e Google che potrebbero utilizzare cookie di profilazione. Nephritic and nephrotic syndromes.
Hyaline and Mucoid degenerations. Membrane receptors and principal pathways in mitogenic signalling. Various types of necrosis. The diapedesis, the chemotaxis, and phagocytosis. Teaching methods Frontal lessons Learning verification modality Oral exam Extended program The inflammation – General characters. Possible educational visits to biotechnological research genersle, which can be planned together with the teacher.
Distinctive characters between acute and chronic phlogosis.
Università degli Studi di Perugia
Diffuse and multinodular goiters, thyroiditis. People search Search with a name Search with a name. Mutation of receptor proteins and transport systems: Main knowledge acquired will be: Intrinsic and Extrinsic Causes of Disease. Diapedesis, chemotaxis and phagocytosis. Vacuolar and hydropic degeneration. Oral examination or, upon request and for special needs of the student to be evaluated with the teacher, case by case, also written test possibilities.
Interstitial nephritis, pyelonephritis and cystitis. Heterogenetic interaction between genes and their products, interaction genes-environment, dynamic and somatic mutations, epigenetic modification.
Outcome of necrotic process. The lectures deal with the whole program of the course of study. Invasive cancer and metastasis. The cells of chronic inflammation. Regressive and Progressive processes. Se procedi nell’utilizzo del Portale accetti l’utilizzo dei cookie presenti. Extended program Pathophysiology of the respiratory system: Metabolism of amino acids: The basic knowledge of these materials are essential geerale the student to understand the concepts etiopathogenetic of molecular and cellular pathology.